A síndrome de Legg-Calvé-Perthes ou doença de Perthes é uma doença degenerativa da articulação da anca (quadril em português brasileiro) em que ocorre. El absceso del psoas es una enfermedad infrecuente en niños. Puede tener una en pediatría es primario; sin embargo, en ocasiones, puede ser de origen secundario y médula espinal, enfermedad de. Legg-Calvé-Perthes, entre otros Download Citation on ResearchGate | Enfermedad de Legg Calvé Perthes (LCP) en varios aspectos de la enfermedad de Legg Calvé Perthes y la evolución de caderas de August · Anales espanoles de pediatria.
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Síndrome de Legg-Calvé-Perthes
J Pediatr Orthop, 19pp. Necesidades del alumnado con diabetes tipo 1 en Espectrophotometric solid-phase tissue plasminogen activator activity assay Sofia-tPA for high-fibrin-affinity tissue plasminogen activators. Conclusions Epidemiological and laboratory data perthess this group ofpediatric patients and from the control group do not supportthe hypothesis that a thrombogenesis defect couldbe the underlying cause of avascular necrosis of the hipjoint. Am J Clin Dee, 94pp.
Anal Biochem,pp. The Kinston antiphospholipid group.
Enfermedad de Legg-Calve-Perthes | HCA Healthcare
Thromb Haemost, 78pp. Blood, 87pp. Identification of the same factor V gene mutation in 47 of 50 thrombosis-prone families with inherited resistance to activated protein C. Thromb Res, 11pp.
Familial hypofibrinolysis and venous thrombosis. Pathophysiology of osteonecrosis of enfermfdad jaw: J Med,pp. The second international anticardiolipin standardization workshop.
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Determination of perfhes activator and its fast inhibitor in plasma. Anticoagulant protein C pathway defective in majority of thromboembolic patients. Clin Orthop,pp. Decreased fibrynolitic potential in patients with idio-pathic avascular necrosis and transient osteoporosis of the hip. Nature,pp.
J Lab Clin Med,pp. Thromb Haemost, 62pp.
Clin Chem, 32pp. Complete study of hemostasis with coagulation and antithrombinfactors as well as study of thrombophilia andfibrinolysis in these patients could determine the prevalenceof hemostasis and fibrinolysis in this group of patients. Are you a health professional able to prescribe or dispense drugs? J Clin Invest, 94pp. The remaining patients were considered withinthe normal range when age was taken into account.
Variability of thrombosis among homozigous siblings with enfermesad to activated protein C due to an Arg-Gln mutaion in gen for factor V. You can change the settings or obtain more information by clicking here. Patients and method Complete study of hemostasis with coagulation and antithrombinfactors as well as study of thrombophilia andfibrinolysis in these patients could determine the prevalenceof hemostasis and fibrinolysis in this group of patients.
Alterations in hemostasis and thrombosis were detectedin one patient who had enfernedad protein Sdeficiency. Genetics aspects of Perthes disease: Protein C and S deficiency, thrombofilia and hypofibrinolysis: Thromb Haemost, peiatriapp.
Clin Othop,pp. Results Alterations in hemostasis and thrombosis were detectedin one patient who had moderate-to-light protein Sdeficiency. Mutation in gene coding for factor V and the risk of myocardial infarction, stroke and venous thrombosis in apparently healthy men.
Blood, 84pp. Blood collection in strongly acid citrate anticoagulant used in a study of dietary influence of basal Tpa activity.
J Biol Chem,pp. Lancet,pp. N Engl J Med,pp. Blood, 82pp.
Association of antithrombotic factor deficiencies and hy-pofibrinolysis with Legg-Perthes disease.